The immunohistochemical studies demonstrated that the majority of tumor cells were immunoreactive for neuroendocrine markers that included chromogranin A, synaptophysin and neuronspecific enolase.
The results indicated that VE had anti-actions on the endocritic function and a decrease in the progesterone level and the ova number in mice, caused by TCDD.
Participants: The Task Force comprised a chair, selected by the Clinical Guidelines Subcommittee (CGS) of The Endocrine Society, six additional experts, one methodologist, and a medical writer.