So combining IgG or IgM, a positive on either and looking at just 20 days or more of having illness you can see that only one test actually captured everybody, that was the test from BioPerfectus.

此结合IgG或IgM，任何一个呈现阳性并且只看20天及以上患病情况，你会发现只有一个测试真正检测出了每个病例，那就是来自BioPerfectus（江苏硕世）测试。

When the abnormal IgA1-IgG immune complexes deposit in the mesangium, the alternative complement pathway is activated, leading to the release of proinflammatory cytokines and migration of macrophages into the kidney, all of which contributes to glomerular injury.

当异常IgA1-IgG免疫复合物沉积在系膜上时，补路途径被激，导致促炎释放，同时促进巨噬细胞向肾迁移，以上所有反应会导致肾小球损伤。

These centroblasts have a rearrangement of their immunoglobulin genes, and some of them undergo a class switch where they change from producing IgM antibodies to producing IgG or IgA antibodies.

这部分中心母细胞免疫球蛋白基发生了重排，其中一些会经历Ig类别转换，从产生IgM变成产生IgG或IgA。

In these sorts of infections, IgA1 antibody production gets ramped up, leading to IgG anti-glycan formation, and immune complex deposition in the glomerular mesangium, causing inflammation.

在这些感染情况下，IgA1抗产生增多，导致抗糖基化抗IgG形成，免疫复合物便沉积在肾小球系膜，产生炎症反应。

And these are known as anti-glycan IgG antibodies.

这些就是抗糖基化IgG抗。

When these anti-glycan IgG antibodies bind to abnormal IgA1, immune complexes are formed.

当这些抗糖基化IgG抗与异常IgA1结合，形成免疫复合物。

In response, the body generates IgG antibodies that target the abnormally glycosylated residues.

在免疫应答下，机产生针对异常糖基化残基IgG抗。

OK, so the B cells make the targeted missiles, the IgG antibodies.

好，B细胞制造了针对目标导弹，也就是IgG抗。

They found that the IgG antibodies present in that SRCC serum specifically bound to the actual cancer cells from the tumors.

他们发现，该SRCC血清中IgG抗能够特异性地结合到肿瘤实际癌细胞上。

Myeloid cells, especially those with CD16, were much better at gobbling up tumor cells when the patient's serum, full of those IgG antibodies, was present.

髓系细胞，尤其是带有CD16细胞，在患者血清（富含IgG抗）存在时，吞噬肿瘤细胞能力显着增强。