A younger organism would naturally release more cytokines than and elderly one.

年轻的有机体自然会比年老的有机体释放更多的细胞因子。

IL-5, on the other hand, activates eosinophils which promote an immune response by releasing more cytokines and leukotrienes.

另方面，IL-5激活嗜酸性粒细胞，通过释放更多的细胞因子和白三烯来促进应。

The release of cytokines causes symptoms like fever, drenching night sweats, and weight loss.

细胞因子的释放会导致发烧、夜间盗汗和体重减轻等症状。

These helper T-cells produce small signaling molecules called cytokines, which activate B-cells and macrophages.

辅助T细胞会产生些小的信号分子，即细胞因子，激活B细胞和巨噬细胞。

They cytokines also attract B-cells and macrophages as part of the inflammatory reaction.

细胞因子也会趋化B巴细胞和巨噬细胞，这也是炎症应的部分。

Known for its antibacterial activity, it increases the release of inflammatory cytokines, which signal cells to repair themselves.

众所周知，它具有抗菌活性，能增加炎症细胞因子的释放，而炎症细胞因子是细胞自我修复的信号。

Along with an increase in proteins called cytokines—which the researchers say might suggest a boost in immune activity.

种名为细胞因子的蛋白质含量升高，对此研究人员表示这可能表明活性提升。

In response, the injured cells release inflammatory molecules called cytokines that activate the immune system to repair the injury.

对此应，损伤的细胞释放出 称为“细胞因子”的发炎分子用来激活系统和修复损伤。

They communicate mostly via tiny information proteins called cytokines.

它们主要通过称为细胞因子的微小信息蛋白进行通讯。

Give it just enough blood to stop the cytokines, but not so much that he bleeds to death.

给它足够的血液来阻止细胞因子的产生，但不要太多，以他失血过多而死。

If the T cell gets activated, it kicks into action, producing cytokines which helps to get other immune cells involved.

T细胞在被激活后，开始合成分泌细胞因子，进而调动其他更多的细胞。

The virus also creates a cytokine storm,   which is when the body releases too many cytokines at once.

该病毒还会产生细胞因子风暴，即身体次释放过多的细胞因子。

This leads to the flood of enzymes and degradation seen in osteoarthritis, but is driven by different inflammatory proteins called cytokines.

也出现骨关节炎中产生大量酶和削弱关节的现象，但由不的炎症蛋白引起，这种蛋白称为细胞因子。

In chronic disease states, chemical messengers called cytokines mediate this pathologic process in the kidney, immune system, and the GI tract.

在慢性疾病中，称为细胞因子的化学信使在肾脏、系统和胃肠道中介导了这病理过程。

In rats and people, intentionally activating the immune system and triggering the release of cytokines increases anxiety.

在老鼠和人身上， 有意激活系统并触发细胞因子的释放会增加焦虑。

And what we find is that inflammatory molecules such as cytokines are increased in response to the diet.

我们发现，炎症分子如细胞因子会随着饮食而增加。

Alcohol also contributes to pancreatitis in other ways, though, for example, stimulating acinar cells to release inflammatory cytokines which attracts a strong immune reaction.

酒精还可以通过其它方式导致胰腺炎，比如促进腺泡细胞释放炎性细胞因子，从而引起强烈的应。

In some cases, the nucleus macrophages attack the nucleus pulposus, and start secreting inflammatory cytokines, like tumor necrosis factor alpha, an interleukin-1, and interleukin-6.

在某些情况下，巨噬细胞会攻击髓核，并分泌炎性因子，比如肿瘤坏死因子α（TNF-α）、白细胞介素-1（IL-1）和白细胞介素-6（IL-6）。

Afterwards, the macrophages release protein molecules called cytokines whose job is to recruit and organize more virus-busting cells from your immune system.

然后巨噬细胞会释放种叫作细胞因子的蛋白质分子它会从系统中征集并组织更多的细胞来杀死病毒。

Soon enough, the handful of white blood cells surveilling the CSF identify the pathogen and release cytokines to recruit additional immune cells.

之后很快，些负责监视脑脊液的白细胞识别出病原体，并释放细胞因子召集额外的细胞。