In the case of the Spanish flu, the cells of infected younger patients released a deleterious excess of cytokines, leading to excessive inflammation of the lungs, and more importantly, tissue necrosis.

以西班牙流感为例，受感染的年轻患者的了过量的有害，导致肺部炎症过重，更重要的是组织坏死。

And most likely it's happening because you are having all of these cytokines and inflammatory markers increasing disproportionately at the end of pregnancy, causing your body to react, almost like it's an autoimmune response.

这很可能是由于体内和炎症标志物在怀孕后期不成比例地增加，导致身体产生类似自身免疫反应的反应。

As " type A" cells in the synovium attempt to remove the debris, immune cells like lymphocytes and macrophages are recruited into the synovial membrane, which produces proinflammatory cytokines that ultimately cause inflammation of the synovium as well, called synovitis.

滑膜上的A型努力清除这些碎片的同时，免疫（如淋巴、巨噬）也被募集进入滑膜，产生炎症，最终导致滑膜炎。

What can happen with asthma is allergens from environmental triggers, like cigarette smoke, are picked up by dendritic cells which present them to a Th2 cell which produce cytokines like IL-4 and IL-5 leading to a number of features of asthma.

哮喘是由环引发的过敏原引起的，比如香烟烟雾，是由树突提呈到Th2中，产生IL-4和IL-5，从而导致哮喘的许多特征。

When the abnormal IgA1-IgG immune complexes deposit in the mesangium, the alternative complement pathway is activated, leading to the release of proinflammatory cytokines and migration of macrophages into the kidney, all of which contributes to glomerular injury.

当异常的IgA1-IgG免疫复合物沉积在系膜上时，补体旁路途径被激活，导致促炎的，同时促进巨噬向肾迁移，以上所有反应会导致肾小球的损伤。

At that point T helper cells, or Th1 cells swoop in and release cytokines —which are cell signaling molecules— like interferon-gamma, and tumor necrosis factor alpha, which further stimulate the inflammatory response.

在这时，辅助T（Th1）突然加入到炎症反应中并；这些是间信号分，包括γ干扰素和肿瘤坏死α等；这些可以进一步促发炎症反应。

Chronic treatment for multiple sclerosis includes immunosuppressants like recombinant beta-IFN which decreases the level of inflammatory cytokines in the brain and increases the function of T regulatory cells.

疾病调节治疗可用免疫抑制剂（重组β-干扰素），可降低大脑中炎性水平和增加调节性T淋巴的功能。

At the same time, the macrophages produce molecules called cytokines and chemokines that alert the rest of the immune system to the presence of the invader.

与此同时，巨噬产生称为和趋化的分，提醒免疫系统的其他部分注意入侵者的存在。

Since they are previously exposed, the immune system's memory t-cells quickly release cytokines to try and control the new outbreak which forms more areas of caseous necrosis.

为宿主之前曾经接触过分枝杆菌，免疫系统中的记忆T快速来试图控制住新一轮疾病的暴发，而这个过程会导致更多的肺部组织形成干酪样坏死。

In response to hypoxia, myocardial cells release signalling molecules called cytokines, which cause dilation of the segment of coronary artery B beyond the blockage.

为了应对缺氧，心肌会这类信号分，使冠脉B阻塞处远端的血管扩张。

In chronic disease states, chemical messengers called cytokines mediate this pathologic process in the kidney, immune system, and the GI tract.

在慢性疾病中，称为的化学信使在肾脏、免疫系统和胃肠道中介导了这一病理过程。

In response, the injured cells release inflammatory molecules called cytokines that activate the immune system to repair the injury.

对此反应，损伤的出 称为“”的发炎分用来激活免疫系统和修复损伤。

Along with an increase in proteins called cytokines—which the researchers say might suggest a boost in immune activity.

同时一种名为的蛋白质含量升高，对此研究人员表示这可能表明免疫活性提升。

Known for its antibacterial activity, it increases the release of inflammatory cytokines, which signal cells to repair themselves.

众所周知，它具有抗菌活性，能增加炎症的，而炎症是自我修复的信号。

When you're asleep, your body releases infection-fighting substances, like antibodies and cytokines, which combat bacteria and viruses.

当你睡觉时，你的身体会抗感染物质，如抗体和，它们可以对抗菌和病毒。

A younger organism would naturally release more cytokines than and elderly one.

年轻的有机体自然会比年老的有机体更多的。

The release of cytokines causes symptoms like fever, drenching night sweats, and weight loss.

的会导致发烧、夜间盗汗和体重减轻等症状。

The cytokines attract Inflammatory cells like macrophages which start releasing even more inflammatory substances like proteases, platelet activating factor, and free radicals, all which contribute to inflammation.

这些可以募集更多的炎症如巨噬；这些巨噬进而更多的炎症介质，如蛋白酶、血小板活化和自由基，它们都可以进一步促进炎症反应。

Newer therapies called biologics can also be useful, because they block the actions of cytokines like tumor necrosis factor alpha and the interleukins.

生物制剂治疗这种新疗法同样有效，能阻断TNF-α和IL-1等的作用。

Soon enough, the handful of white blood cells surveilling the CSF identify the pathogen and release cytokines to recruit additional immune cells.

之后很快，一些负责监视脑脊液的白识别出病原体，并召集额外的免疫。