HOWEVER, a new study out of Germany finds that high-intensity exercise puts us at risk for more than just cardiac hypertrophy.

然而，德国一项新研究显示，高强度运动不只将我们暴露在脏肥风险中。

Compensatory hypertrophy of Turbinates occur in other side.

另一侧出现代偿性鼻甲肥。

Whatever the irritants are, their effect is to stimulate hypertrophy and hyperplasia of the mucinous glands in the main bronchi, as well as the goblet cells in the smaller airways - the bronchioles, which increases mucus production in both locations.

无论哪种刺激，都会激发主支气管黏液腺以及小气道杯状细胞的肥增生，使得肺内各个部位黏液产生增加。

So in response, the right ventricle can hypertrophy, or get larger, and the left might atrophy, or get smaller, and these massive changes in heart structure can ultimately cause the heart to fail.

所以相应地，右会肥，会萎缩变小，并且这些脏结构的变化会最终导致脏衰竭。

They do this through beta cell hyperplasia, an increased number of beta cells, and beta cell hypertrophy, where they actually grow in size, all in this attempt to pump out more insulin.

它们通过β细胞增生，即增加β细胞的数量，以及β细胞肥，即其体积增，泵出更多的胰岛素。

Essentially, Greg argues that classifications require proper context of the data being interpreted, especially in something like hypertrophy, where small effect sizes are generally observed.

Greg 认为，效应量分类需要结合具体数据的背景来解读，尤其是在肌肉增长研究中，小的效应量本身就很常见。

Normally, this ratio should be less than 40%, but it can be over 40% for people with chronic bronchitis, because of the hyperplasia and hypertrophy of the glands.

一般情况下应少于40%，对于慢支患者来说，由于黏液腺的肥增生，该数值会于40%。

With this in mind using his super fitness science, A new range of effect size classifications that might be more fitting for muscle hypertrophy outcomes.

基于这一点，他利用自己的健身科学知识，提出了一种可能更适用于肌肉增长研究的效应量分类标准。

Well that hypertrophy is concentric, which means that the new sarcomeres are generated in parallel with existing ones.

肥是向性的，这意味着有新肌节产生，与之前的肌肉平行生长。

When chronic lung disease leads to right-sided hypertrophy and failure, it's known as cor pulmonale.

当慢性肺病导致右侧脏肥和衰时，被称作肺病。

And, when we look back at the muscle research, there does seem to be a consistent result, just not what the hypertrophy rangers hoped for.

而且，当我们回顾肌肉研究时，似乎确实有一个一致的结果，只是不是增厚拥护者所希望的。

Just like how the soup is not too cold or too hot… the hypertrophy range is juuuuust right, at least for Brodilocks.

就像汤不太冷也不太热一样......肌肉增厚的范围是正确的，至少对布罗迪洛克来说是这样。

To compensate, the left ventricle actually bulks up, and its muscles hypertrophy, or grow so that the ventricle can contract with more force.

为了代偿，会胀，它的肌会过度生长而变得肥让能更力收缩。

Concentric hypertrophy leading to diastolic failure can also be caused by aortic stenosis, which is a narrowing of the aortic valve opening, as well by hypertrophic cardiomyopathy, an abnormal ventricular wall thickening often from a genetic cause.

导致舒张性衰的向性肥，也可能由主动脉瓣狭窄（即主动脉瓣开口变窄）造成，也可能由肥厚型肌病（一种遗传性异常壁增厚）引起。

Wherever we seem to look though, the data makes it abundantly clear that there is no clear physiological hypertrophy advantage to any specific rep range.

然而，无论我们分析哪里的研究，数据都非常清楚地表明，任何特定的数据范围都没有明显的生理增厚优势。

This idea is rooted from claims that muscle hypertrophy, or muscle growth, is best achieved with short inter-set rest intervals of about 30 to 60 seconds.

这一观点源于一个说法，那就是肌肉生长最好通过约 30 到 60 秒的短间歇时间来实现。

This, just like before, makes it harder for the right side of the heart to pump against and can lead to right-sided hypertrophy and heart failure.

如前述提及，这会让脏右侧更难向肺部泵入血液，并可能导致右侧脏肥和力衰竭。

That's power, strength, cardio and conditioning, mobility, hypertrophy, and athleticism.

那是力量、强度、肺功能和体能训练、灵活性、肌肉肥以及运动能力。

This is where the hypertrophy range makes its triumphant return.

这就是增厚范围优胜的地方。

In hypertension, remember how the left ventricular hypertrophied?

在高血压的情况下，还记得是如何变肥的吧？