Objective To analyze the aetiological characteristics and bacterial susceptibility in patients with ventilator-associated pneumonia (VAP) .
目的分析呼吸机相关肺炎(VAP)的病原学以及药
敏感情况。
Here, we show that deletions of VPS28 and VPS32 increase the susceptibility of C. albicans to cell wall disruption agents, echinocandin and azole antifungal agents.
【文献摘要】Vps28p and Vps32p act in both the endocytic and the pH signaling pathways in yeasts and are required for Candida albicans virulence.
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