Shock after right ventricular MI will usually respond partially to volume expansion;however, vasopressor agents may be needed.
右MI克通常会对容量扩张反应,但可能需要血管。
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So the mechanism, it is alpha-1 agonist causing vasopressor constriction, beta-1 agonist causing increased cardiac output.
其作用机制是α-1动引起血管收缩,β-1动导致心输出量增加。
So norepinephrine, tried-and-true vasopressor, right, we use in the ICU all the time, very potent, has good evidence, decent evidence, I would say, in HRS.
去甲肾上腺素,种经过时间考验的强效血管收缩剂,ICU中经常使用,效果非常显着,肝肾综合征(HRS)中的证据也较为充分,认为证据相当可靠。
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