Coagulase-positive bacteria, like Staph aureus, convert the soluble fibrinogen into sticky fibrin, which then visibly clumps up.

凝固酶阳性细菌（如金黄色葡萄球菌）能将可溶性纤维蛋白原转化为粘性纤维蛋白，从而出肉眼可见团聚。

The final step is activation of the protein fibrinogen to fibrin, which deposits and polymerizes to form a mesh around the platelets.

最后一步是纤维蛋白原活化为纤维蛋白，聚集网包绕着血小板。

This gel-like tissue contains bundles of collagen, called “fibrils.”

种凝胶状组织含有胶原蛋白束， 称为“原纤维” 。

Some of these aggregate into large insoluble fibrils that deposit in the brain as plaques.

其中一些聚集不溶性原纤维，以斑块形式沉积在脑中。

And it could also use the like actinofibrils — these structures within the wings that actually allowed it to change the shape a little bit.

它也可以使用类似放线原纤维东西——翅膀内些结构实际上允许它稍微改变形状。

The final reaction is the conversion of fibrinogen to threads of fibrin that trap blood cells and plasma to produce the clot.

最后反应是将纤维蛋白原转化为纤维蛋白，捕获血细胞和血浆以产生凝块。

Thrombin then turns factor I or fibrinogen, which is soluble, into factor Ia or fibrin, which is insoluble and precipitates out of the blood at the site of injury.

之后凝血酶将可溶因子I（纤维蛋白原）转变为因子Ia（纤维蛋白原），纤维蛋白不可溶，便在损伤部位从血液中析出。

They evade threats and launch counter-attacks using their mutable collagenous tissue, or MCT. This gel-like tissue contains bundles of collagen, called " fibrils." Proteins can interact with these fibrils to slide them together, stiffening the tissue, or apart, softening it.

他们使用可变胶原组织 (MCT) 躲避威胁并发动反击。 种凝胶状组织含有束胶原蛋白， 称为“原纤维” 。蛋白质可以与些原纤维相互作用， 使它们滑动在一起， 使组织变硬， 或分开，使其软化。