When pathogens enter the lungs, the macrophages work to destroy them.

当病因抵达肺部，巨噬细就会开始摧毁它们。

It has the ability to stop phagolysosomal fusion, and survive and replicate inside alveolar macrophages.

它可以阻止吞噬体和溶酶体融合，从而在肺泡巨噬细内生存并增殖。

In this stage the exudate gets digested by enzymes, ingested by macrophages, or coughed up.

在个阶，物被酶解，由巨噬细吞噬或咳出。

They cytokines also attract B-cells and macrophages as part of the inflammatory reaction.

细因子也会趋化B淋巴细和巨噬细，也是炎症反应的一部。

Once the macrophages recognize the bacteria, they can then go on to destroy by eating it.

一旦巨噬细识别出细菌，它们就会通过吃掉它来杀死它。

Both macrophages and neutrophils are part of the so-called Innate Immune System.

巨噬细和中性粒细都是所谓的先天免疫系统的一部。

If so, mycobacterium tuberculosis will reproduce inside those macrophages, and form colonies in the surrounding lung tissue.

核杆菌会在巨噬细内繁殖，并在周边组织中形成菌落。

And then they're then called " hemosiderin-laden macrophages" , also known as " heart failure cells" .

因而他们被称为“含铁血黄素巨噬细”，也称作“心力衰竭细”。

First, specialized cells called macrophages eat the invading material in an attempt to clean up the inflammatory mess.

首先，巨噬细会吃掉外来物质，试图清理有可能出现的炎症。

Some monocytes migrate into tissues and differentiate into macrophages, which remain in tissues and aren't found in the blood.

一些单核细迁移到组织中，化成为巨噬细；它们会停留在组织里，不会再在血液里出现。

Here, immune cells called macrophages rush to the infection site, attempting to absorb and break down the bacterial invaders.

肺里的巨噬细会赶到受感染处做出免疫反应，努力吞噬、解些细菌侵略者。

Cells called macrophages summon all sorts of others to an injury, to try to repair it.

巨噬细召集其他免疫细到达受伤处，并试图修复伤害。

These helper T-cells produce small signaling molecules called cytokines, which activate B-cells and macrophages.

辅助T细会产生一些小的信号子，即细因子，激活B细和巨噬细。

The laser beam breaks the ink globules into smaller particles that can then be cleared away by the macrophages.

激光把颜料块击碎，变成更小的颗粒，巨噬细可以将其清除。

It doesn't just survive though, it proliferates and creates a localized infection.

并且它的目的并不仅仅是生存下去，核枝杆菌在巨噬细内繁殖造成局部感染。

But macrophage signalling also involves SNAP-25, so Dr Chapman thinks he can use botulinum toxin to shut the cells up.

但是巨噬细的信号通路中也包括SNAP-25，因此查普曼博士想，他能够用肉毒素来关闭些免疫细。

This means that it protects the bacteria against phagocytosis by macrophages and neutrophils, allowing Klebsiella to escape destruction.

它可以保护细菌免受巨噬细和中性粒细的吞噬，使克雷伯杆菌不能被消灭。

First, phagocytes trigger the immune response by sending macrophages and dendritic cells into the blood.

首先，吞噬细通过将巨噬细和树突状细送入血液来启动免疫反应。

Cytokines are then released, activating cytotoxic T cells which, along with macrophages, destroy infected cells.

随后，细因子被释放， 激活了杀伤性T细，些T细与巨噬细协同作用， 摧毁受感染的细。

Macrophages are part of the immune system.

巨噬细是免疫系统的一部。