Within 48 hours, you could have a condition called pulmonary edema.

48小时内，你可能会出现肺肿的症状。

People do get high altitude pulmonary edema and high altitude cerebral edema here as well.

这里也会发生高原肺肿和高原脑肿。

See? See, his b. u. n. and creatinine are increasing. He clearly has pulmonary edema.

看到了吗？他已经 BUN了 (血液尿素氮化)。还有肌氨酸酐正在增加。很明显他有肺肿。

Pulmonary edema can develop in a few ways and often develops through a combination of mechanisms.

肺肿的发展可以有好几种方式，而且往往通过多种机制的结合发展。

Regardless of the cause, pulmonary edema interferes with gas exchange and results in shortness of breath.

无论病因是什么，肺肿会干扰肺，并导致短促。

Treatment for pulmonary edema typically involves giving supplemental oxygen.

肺肿的治疗通常包括吸氧。

But knowing that if we didn't warm ourselves that night, we would die from pulmonary edema.

如果我们那晚不保暖，就会因肺肿而死。

But it can have longer term effects: headache, nausea, disorientation, dizziness, pulmonary edema, death.

它可能会产生长期影响：头痛、恶心、迷失方向、头晕、肺肿、死亡。

If the cause is related to inflammation or low oncotic pressure, then managing that illness will help resolve the pulmonary edema.

如果病因与炎症反应或低胶体渗透压有关，那么治疗潜在疾病有助于治疗肺肿。

The diagnosis of pulmonary edema is usually made with a chest x-ray or chest CT scan that shows fluid in the interstitial space.

肺肿常通过X线胸片或胸CT显示肺间质淤血诊断。

Once again, blood starts to back up in the left atrium, pulmonary veins, and pulmonary capillaries, ultimately leading to pulmonary hypertension and pulmonary edema.

血液又一次在左心房、肺静脉、肺毛细血管淤积，最终导致肺高压和肺肿。

In the acute onset of AFib, if there is significant hemodynamic compromise, pulmonary edema, or evidence of coronary ischemia, emergent cardioversion is recommended.

在心房颤动（AFib）急性发作时，若出现显着的血流动力学损害、肺肿或冠状动脉缺血的证据，建议立即进行心脏电复律。

Pulmonary edema can lead to severe shortness of breath, and in left-sided heart failure, it can lead to orthopnea which is when there's worse shortness of breath while lying flat.

肺肿会导致短，左心衰时可出现端坐呼吸，即平躺时呼吸更加困难。

Nene, who heads the institute’s Vaccine Biosciences Program, said East Coast fever can eventually cause pulmonary edema -- a potentially fatal build-up of fluid in the lungs.

内内， 作为研究所疫苗生物科学项目的负责人，表示东海岸热最终可能导致肺肿——这是一种可能致命的肺液体积聚。

Regardless of the cause, low oncotic pressure leads to fluid moving from the capillary and into the interstitial space throughout the body, and in the lungs that results in pulmonary edema.

无论何种原因，低胶体渗透压导致血浆从毛细血管进入全身组织间隙，在肺中便导致肺肿。

To understand pulmonary edema, let's first talk about the three main factors that determine how fluid moves between the capillaries and interstitial fluid, which are the hydrostatic pressure, oncotic pressure and capillary permeability.

为了解肺肿，我们首先讲讲决定液体如何在毛细血管和间隙之间移动的3个主要因素：1.流体静压，2.胶体渗透压，3.毛细血管通透性。

Pulmonary edema makes gas exchange difficult because oxygen and carbon dioxide have to diffuse through a wide layer of interstitial fluid, to get from the alveoli to the pulmonary capillary and vice versa.

肺肿导致肺障碍，因为氧和二氧化碳须透过较厚的间隙液体层，从而在肺泡和肺毛细血管间交。

Alright, as a quick recap, pulmonary edema refers to fluid accumulation in the interstitial space of the lungs which can be seen on a chest Xray or chest CT scan.

好！快速回顾一下，肺肿是指X线胸片或胸CT可见肺组织间隙积液。

Now, the underlying cause of pulmonary edema can be cardiogenic - meaning that it develops as a result of a heart disease, or can be non-cardiogenic which typically involves damage to the pulmonary capillaries or alveoli.

肺肿的根本原因可能是心源性的，意味着这可能是心脏疾病造成的，也可能是非心源性的，与肺毛细血管或肺泡受损有关。

A backup of blood in the pulmonary veins and capillary beds can increase the pressure in the pulmonary artery and can also result in fluid moving from the blood vessels to the interstitial space causing pulmonary edema, or congestion.

而血液在肺静脉和毛细血管的积留，会提升肺动脉的压力，还会导致体液从血管流入组织间隙，引起肺肿。