They basically are the endothelium, the inside lining of the blood vessel floating around.

它们基内皮，漂浮在周围的的内层。

And the endothelium of these capillaries is very porous.

这些毛细的内皮细多孔性很强。

Cortisol can also cause the endothelium, or inner lining of blood vessels, to not function normally.

压力荷尔蒙皮质醇会令你的内循环或中的内覆组织无法正常运行。

The first way is called the extrinsic pathway, which starts when tissue factor gets exposed by the injury of the endothelium.

第一种外源性凝途径由组织因子暴露于壁损伤处后启动。

It also releases Sphingomyelinase C toxin, which destroys red blood cells as well as the endothelium of the capillaries, causing hemorrhage.

它也能释放出鞘磷脂酶C，破坏红细和内皮细，从而导致出。

So that means, so nitric oxide is a gas, it's produced in the endothelium.

所以这意味着，氮氧化物一种气体，它在内皮中产生。

Well, LDL cholesterol can penetrate the endothelium.

嗯，低密度脂蛋白胆固醇可以穿透内皮。

Even large LDL can penetrate the endothelium.

即使较大的 LDL 也能穿透内皮。

So the endothelium is the single layer of cells that line every blood vessel throughout the body.

因此，内皮遍布全身每条壁的单层细。

Now, small oxidized penetrates more easily, but it carries less total cholesterol and deposits less cholesterol in the endothelium.

现在， 小的氧化物更容易渗透，但它携带的总胆固醇较少， 并且在内皮中沉积的胆固醇也较少。

During stasis, platelets and other clotting factors contact the endothelium, and prolonged interaction leads to clotting factor adhesion, and, ultimately, activation of the clotting cascade.

流瘀滞发生时，小板、其他凝因子、内皮细长时间的互相作用，最终导致凝级联反应的激活。

If you wanna apply the same logic of what we have this, and LDL, by the way, can cause inflammation in the endothelium.

如果你想应用与我们相同的逻辑，顺便说一下， LDL 会导致内皮细发炎。

After that, some platelets adhere to the damaged vessel wall, and become activated by collagen and tissue factor, proteins which are normally kept separated from the blood by an intact endothelium.

接着，小板粘附至损伤的壁，并且被胶原和组织因子活化；这些蛋白质平常由完好的壁与液隔离。

Finally, there's the innermost layer of the heart called the endocardium, which is made of a relatively thin layer of endothelium which is the same layer of cells that line the blood vessels.

最后，心脏的最内层叫心内膜，由一层较薄的内皮细构成；与位在内壁的同一层细。

Normally, after a cut and damage to the endothelium, or inner lining of blood vessel walls, there's an immediate vasoconstriction or narrowing of the blood vessel which limits the amount of blood flow.

正常情况下，壁内层的内皮细受到损伤后，会立刻收缩变窄，减少流量。

The cool thing about this layer is that it contains the endothelium, which you may remember is made up of simple squamous epithelium tissue and is continuous with the lining of the heart.

这层最酷的地方它包含内皮，你可能记得内皮由简单的鳞状皮组织组成的，并且与心脏内膜连续。

Normally, the process starts with damage to the endothelium, or inner lining of blood vessel walls, after which there's an immediate vasoconstriction or narrowing of the blood vessel which limits the amount of blood flow.

通常，这一过程开始于内皮损伤，之后立即发生收缩，或者说通过收缩来限制流量。

The moment the endothelium, that is the inner lining of the red blood vessel, when it feels injury, it sends a whole cascade of markers and signals and say, " Yo, I'm like, I'm cut here."

那一刻内皮，也就红色的内层，它感觉到受伤时，它会发送一连串的标记和信号说，“哟，我就像，我这里被割伤了。”