That's why we prefer the more slow-burn feeling we get when our brains release acetylcholine.

这也是为什么我们比较喜欢脑中释放乙酰胆碱时平稳和缓的感受。

One type of medication is an acetylcholinesterase inhibitor, like neostigmine or pyridostigmine.

其中一种药物是乙酰胆碱酯酶（AChE）抑制，如新斯的明或吡斯的明。

In the central nervous system, nicotinic receptors are on pre-synaptic axon terminals of neurons, and when nicotine binds to them, it triggers the release of neurotransmitters like dopamine, acetylcholine, and glutamate, which is why it's considered an acetylcholine agonist.

在中枢神中，烟碱受体分布于突触前神元的轴突末端，当尼古丁与受体结合后，会触发多巴胺、乙酰胆碱、谷氨酸盐等神递质的释放，因此尼古丁被认为是一种乙酰胆碱受体。

For now you learned that your sympathetic nervous system controls your body's stress response and how the signals in your sympathetic nervous system travel to an effector, using the neurotransmitters acetylcholine in the ganglion and norepinephrine at the effector.

现在知道了的交感神控制身体的压力反应，以及交感神中的信号如何传递到效应物，使用神节中的神递质乙酰胆碱和效应物处的去甲肾上腺素。

Symptoms of Schizophrenia appear when one of the neural circuit situated in the midbrain, called Substantia nigra, overproduce neurotransmitter called Dopamine and disrupts the balance between secretion of Dopamine and Acetylcholine.

当位于中脑的黑质神回路产生过量多巴胺，并破坏多巴胺和乙酰胆碱分泌之间的平衡时，就会出现精神分裂症的症状。

Even though this might be oversimplifying things a bit, the brain regions affected by HD have decreased GABA and acetylcholine, and increased dopamine levels.

尽管这可能有点过于简单化，但罹患HD患者的大脑内GABA和乙酰胆碱的水平减少，多巴胺水平增高。

That happens when we concentrate, read, or focus our minds.

在我们专注、阅读或集中精神的时候，脑中就会释放乙酰胆碱。

If you haven't heard of acetylcholine yet, you're gonna wanna remember that name.

如果还没听说过乙酰胆碱，会想记住这个名字的。

Once nicotine is absorbed into the bloodstream, it binds to a type of acetylcholine receptor, called a nicotinic acetylcholine receptor - also called a nicotinic receptor - which is found throughout the body and brain.

一旦尼古丁进入血液循环，它就会和一种乙酰胆碱受体结合，也称烟碱型乙酰胆碱受体或烟碱受体；这类受体广泛分布于身体和大脑中。

First you've got your motor neurons that release the neurotransmitter acetylcholine at the neuromuscular junction, which then binds to nicotinic acetylcholine receptors on muscle cell membranes.

首先，的运动神元在神肌肉接头处释放神递质乙酰胆碱（ACh），然后与肌肉细胞膜上的N胆碱受体相结合。

There are two important chemicals found in all our brains - dopamine and acetylcholine.

多巴胺和乙酰胆碱。

So, that acetylcholine crosses the synapse and, if there's enough of it, it can stimulate action potentials in several neurons on the other end — in the postganglionic fibers.

乙酰胆碱穿过突触，如果有足够的乙酰胆碱，它可以刺激另一端的几个神元的动作电位——在节后纤维中。

Since usually there's this balance of signaling between dopamine and acetylcholine, a loss of dopamine reaching the striatum increases the relative amount of acetylcholine signalling there.

由于多巴胺和乙酰胆碱之间有一种平衡，多巴胺减少的信号传到纹状体，导致乙酰胆碱信号发出相应增加。

Your body uses choline to produce a neurotransmitter called acetylcholine, which plays key roles in learning, memory, attention, motivation, and healthy sleep.

的身体使用胆碱产生一种叫做乙酰胆碱的神递质，它在学习、记忆、注意力、动力和健康睡眠中起着关键作用。

In both systems, neurons release acetylcholine, or ACh, in their preganglionic synapses.

在这两个中，神元在节前突触中释放乙酰胆碱。

Motor symptoms respond to drugs used to treat Parkinson's disease, like levodopa, a dopamine analogue, while cognitive symptoms may improve with drugs for Alzheimer's disease, like donepezil, which increases acetylcholine availability.

针对运动障碍，可使用与帕金森病类似的药物，比如多巴胺类似物左旋多巴，而使用类似阿尔茨海默病的药物，可以改善认知症状，比如多奈哌齐，它能增加乙酰胆碱的供给。

The amount of choline in the diet influences how much acetylcholine is available in the brain and central nervous system.

饮食中胆碱的含量会影响大脑和中枢神中乙酰胆碱的含量。

Most neurons in the cerebral cortex are called cholinergic neurons, because they produce acetylcholine.

大脑皮层中的大多数神元为胆碱能神元，因为它们产生乙酰胆碱作为神递质。

Like all preganglionic fibers, these release acetylcholine, too.

像所有节前纤维一样，它们也释放乙酰胆碱。

Acetylcholinesterase degrades acetylcholine, so acetylcholinesterase inhibitors stop acetylcholinesterase from breaking down acetylcholine, which ends up increasing the concentration of acetylcholine around muscle cells and helps counteract the effects of acetylcholine receptor antibodies.

AChE水解乙酰胆碱，所以AChE抑制可以阻止AChE分解乙酰胆碱，从而增加肌肉细胞附近的ACh浓度，减少ACh受体抗体的影响。