This gel-like tissue contains bundles of collagen, called “fibrils.”

这种凝胶状组织含有胶蛋白束， 称为“” 。

Some of these aggregate into large insoluble fibrils that deposit in the brain as plaques.

其中一些聚集成大的不溶性，块形式沉积在大脑中。

Coagulase-positive bacteria, like Staph aureus, convert the soluble fibrinogen into sticky fibrin, which then visibly clumps up.

凝固酶阳性细菌（如金黄色葡萄球菌）能将可溶性蛋白转化为粘性蛋白，从而出现肉眼可见的团聚现象。

And it could also use the like actinofibrils — these structures within the wings that actually allowed it to change the shape a little bit.

它也可使用类似放线的东西——翅膀内的这些结构实际上允许它稍微改变形状。

The final step is activation of the protein fibrinogen to fibrin, which deposits and polymerizes to form a mesh around the platelets.

最后一步是蛋白活化为蛋白，聚集成网包绕着血小板。

The final reaction is the conversion of fibrinogen to threads of fibrin that trap blood cells and plasma to produce the clot.

最后的反应是将蛋白转化为蛋白，捕获血细胞和血浆产生凝块。

Thrombin then turns factor I or fibrinogen, which is soluble, into factor Ia or fibrin, which is insoluble and precipitates out of the blood at the site of injury.

之后凝血酶将可溶的因子I（蛋白）转变为因子Ia（蛋白），蛋白不可溶，便在损伤部位从血液中析出。

They evade threats and launch counter-attacks using their mutable collagenous tissue, or MCT. This gel-like tissue contains bundles of collagen, called " fibrils." Proteins can interact with these fibrils to slide them together, stiffening the tissue, or apart, softening it.

他们使用可变的胶组织 (MCT) 躲避威胁并发动反击。 这种凝胶状组织含有成束的胶蛋白， 称为“” 。蛋白质可与这些相互作用， 使它们滑动在一起， 使组织变硬， 或分开，使其软化。