Some causes of cerebral edema are an large ischemic strokes and meningitis.

脑水肿可能的原因还包括严重脑梗、脑膜炎。

Ischemic and infarcted tissue results in the sloughing off of intestinal mucosal tissue, blood, and mucus into the gut.

及梗使肠粘膜组织脱落，与液、粘液一同进入肠道。

The displaced part of the brain can compress the anterior cerebral artery, and that can cause an ischemic stroke.

大脑移位的部分会压迫大脑前动脉，可导致脑梗。

And infratentorial herniations can compress the vital area in the brain stem that controls consciousness, respiration, and heart rate.

幕上脑疝可能压迫脑的供养动脉，造成脑梗；枕骨大孔疝可能压迫脑干的生命中枢，即控制意识、呼吸和心率的部位。

Another cause is Dressler syndrome which occurs several weeks after a myocardial infarction, or heart attack.

另一个病因是发生于心肌梗数周后的心肌梗后综合征。

STEMI is infarction that extends the entire thickness of the myocardium.

STEMI指心肌梗延伸至心肌全层。

Basically, when heart cells die in a myocardial infarction, it leads to massive inflammation that also involves the serous pericardium.

心肌梗导致心肌亡，引发累及浆膜心包的强烈炎症反应。

Your heart is racing or what we call arrhythmias and other cardiac symptoms can present as myocardial infarctions.

你的心跳加速，或者我们称之为心律不齐，以及其他心脏症状可能表现为心肌梗。

It should be noted, however, that this condition is clinically unstable and often precedes a myocardial infarction.

心肌肌钙蛋白水平保持正常，或只是轻微升高。然而，应该指出的是，这种疾病在临床上是不稳定的，而且常常发生在心肌梗之前。

In severe cases, blood flow can been completely cut off, which results in myocardial infarction, or the necrosis of a portion of myocardium.

严重的情况下，流会被完全阻断，引起心肌梗，即心肌的部分坏。

A prolonged ischemia can lead to death of the heart tissue, known as heart attack or myocardial infarction.

液供应减少称为。长时间可导致心脏组织亡，即心脏病发作或心肌梗。

Okay, let's recap - acute pericarditis is usually caused by a idiopathic causes, but can be caused by viruses or after a myocardial infarction.

急心包炎通常原因未明，也可因病毒感染或心肌梗导致。

However, in a lead looking directly at an acute MI the T waves may flip and become inverted.

然而，直接观察急心肌梗导联，T波可能会翻转并倒置。

In contrast partial occlusion may progress to localized infarction.

相反，部分闭塞可发展为局部梗。

This is an extreme case of clockwise rotation and is suggestive of extensive anterior myocardial infarction.

这是顺时针旋转的极端情况，提示广泛的前壁心肌梗。

The posterior cerebral artery can also be compressed, which results in ischemic stroke of the occipital part of the brain, which is responsible for processing of visual information.

另外，大脑后动脉也会受到压迫，导致大脑枕部脑梗；大脑枕部区域本来负责视觉信息的处理。

Elevation of more than two small squares in the chest leads or one small square in the limb leads, indicates the possibility of myocardial infarction.

胸导联＞2个小方格抬高或肢体导联＞1个小方格抬高，提示可能心肌梗。

The development of pathological Q waves occurs nine to twelve hours after the onset of occlusion or in the days following infarction.

病理Q波的发展发生在闭塞开始后9～12小时或梗后几天。

While ST segment elevation generally resolves within days and T-wave inversion within weeks following the infarction.

而ST段抬高一般在梗后几天内消失，T波倒置一般在梗后几周内消失。

Late pathological Q waves are a permanent marker of such an MI on the patient's subsequent ECGs.

晚期病理Q波是患者随后ECG上心肌梗的永久标志。