First one happens in the liver, where inactive vitamin D is converted into 25-hydroxy-vitamin D by the enzyme 25-Hydroxylase.

首先在肝脏，没有活性的维生素D在25-羟化酶的作用下被转化为25-羟维生素D。

Additionally, parathyroid hormone can boost 1-alpha-hydroxylase activity, which forms more active vitamin D, increasing gut absorption of calcium.

此外，甲状旁腺激素可以增加1-α-羟化酶的活性，从而形成更多有活性的维生素D，增加肠道对钙的吸收。

Hobgood says in her paper that “low activity DBH would relate to impulsive behaviors while high activity DBH would relate to persistent behaviors.”

Hobgood 在文低活性的多巴胺-ß-羟化酶跟冲动行为有关，高活性的多巴胺-ß-羟化酶与常态行为有关。

First, with the help of enzyme tyrosine hydroxylase (TH), tyrosine gets converted to L-dopa, also known as levodopa.

首先，在酪氨酸羟化酶（TH）的帮助下，酪氨酸也转化为左旋多巴被称为左旋多巴。

This is why he recommends type O people to do vigorous exercise and eat a lot of protein because that will block the enzyme DBH.

这就是为什么他推荐 O 型血的人做激烈运动和吃很多蛋白质，因为这会阻断 DBH（多巴胺-ß-羟化酶）。

25-hydroxy-vitamin D then travels to the kidneys, where the enzyme 1-alpha-hydroxylase converts it to 1,25 hydroxy-vitamin D, or calcitriol, which is the active form of vitamin D.

之后25-羟维生素D被运送至肾脏，在1-α-羟化酶的作用下被转化为1，25-二羟维生素D，即骨化三醇，它是维生素D的活性形式。

Medications like phenytoin, an anticonvulsant, uses liver and kidney hydroxylase enzymes for its own metabolism, so especially with long term use, there's not enough vitamin D being generated.

苯妥英，一种抗惊厥药物，会占用肝肾的羟化酶用于药物代谢，所以对于尤其是长期用该药的患者，其维生素D生成可能不足。

He explains that the neurotransmitter dopamine is converted into the neurotransmitter norepinephrine by an enzyme called dopamine beta-hydroxylase, and that blood type O people have higher levels of DBH, so they are prone to anxiety from increased levels of norepinephrine.

他解释神经递质多巴胺通过一种叫做多巴胺-ß-羟化酶的酶转化成神经递质去甲肾上腺素，O 型血的人有高水平的DBH（多巴胺-ß-羟化酶），所以他们容易因去甲肾上腺素的增加而焦虑。