Trypsin is a pancreatic digestive enzyme that is produced as the zymogen trypsinogen.

蛋白酶是一种消酶，以蛋白酶原形式产生。

The level of alpha-1 antitrypsin in the blood can also be measured.

也可测量血清α-1抗蛋白酶水平。

So this antitrypsin protein is a protease inhibitor, and inactivates trypsins, thereby preventing protein breakdown.

而抗蛋白酶就是一种蛋白酶抑制剂，通过抑制蛋白酶防止蛋白质水解。

Zymogen granules might fuse with lysosomes bringing trypsinogen into contact with lysosomal digestive enzymes.

酶原颗粒可能会和溶酶体融合，使蛋白酶原与溶酶体消酶接触。

Cirrhosis caused by alpha-1 antitrypsin can be diagnosed with a liver biopsy.

α-1抗蛋白酶导致的肝硬可通过肝穿活诊断。

Normally, trypsinogen isn't activated until it is cleaved by protease enteropeptidase which is found in the duodenum.

通常情况下，蛋白酶原在被十二指肠中的肠肽酶切割之后才会被激活。

Contemporary, 'normal' flu viruses require the presence of the enzyme trypsin in their human host, in order to replicate.

在当代，“正常”流感病毒需要在其人类宿主中有蛋白酶才能复制。

The 1918 virus instead could proliferate even in the absence of trypsin, making it more pathogenic than other viruses.

1918年的病毒甚至在没有蛋白酶的情况下也能增殖，使得它比其他病毒更具致病性。

This test stains glycoproteins like alpha-1 antitrypsin a pink color.

测将如α-1抗蛋白酶等糖蛋白染成粉红色。

There's also pancreatic amylase which breaks down carbohydrates into shorter oligosaccharides, and proteases, like trypsin, which cleaves proteins down into smaller peptides.

另外还有淀粉酶，可将碳水合物分解为小分子的寡糖；再者还有蛋白酶，如蛋白酶，可将蛋白质分解成小分子肽链。

Symptoms of alpha-1 antitrypsin deficiency typically involve the lungs and liver.

α-1抗蛋白酶缺陷的典型症状累及肺和肝。

Since you need both mutant copies for the disease, this makes alpha-1 antitrypsin deficiency a recessive disease.

由于罹患α-1抗蛋白酶缺陷必须有2突变基因拷贝，α-1抗蛋白酶缺乏症是一种隐形遗传病。

The gene that encodes alpha-1 antitrypsin protein is called SERPINA1, which stands for serpin peptidase inhibitor, clade A, member 1.

编码α-1抗蛋白酶的基因是SERPINA1，代表丝氨酸蛋白酶抑制剂，A系，1成员。

Trypsins are a type of protease, which is an enzyme that can break down other proteins.

蛋白酶是一类蛋白酶，能够水解多种蛋白质。

Another effect of unchecked inflammation in alpha-1 antitrypsin deficiency is chronic bronchitis, resulting from increased mucus production and narrowing of the airways.

α-1抗蛋白酶缺乏症导致炎症失控的另一后果是慢性支气管炎，由黏液增多以及气道狭窄导致。

Trypsinogen might then be turned into activated trypsin which begins the cascade of digestive enzyme activation and autodigestion of the pancreas—which is acute pancreatitis.

于是蛋白酶原可能被转为活的蛋白酶，开启消酶激活的级联反应，导致腺自溶——就是急性腺炎。

Diagnosis of alpha-1 antitrypsin deficiency often begins with a chest X-ray or Chest CT to look for hyperinflated lungs or evidence of damaged lung tissue.

α-1抗蛋白酶缺乏症的诊断常通过X线胸片或胸部CT寻找肺过度膨胀或肺组织受损的证据。

So alpha-1 antitrypsin deficiency can lead to both emphysema and chronic bronchitis, the two types of chronic obstructive pulmonary disease, or COPD.

因此α-1抗蛋白酶缺乏症可导致肺气肿和慢支，而肺气肿和慢支是慢阻肺（COPD）中的2种类型。

You've got trypsin and peptidase in there, which break proteins down into amino acids, and you have lipases that turn triglycerides into fatty acids and glycerol.

你有蛋白酶和肽酶，它们将蛋白质分解成氨基酸，还有脂肪酶将甘油三酯转为脂肪酸和甘油。

To digest a meal, these zymogens are released into the pancreatic duct, and delivered to the small intestine where they are activated by the protease trypsin.

当消食物时，些酶原被释放入管，然后进入小肠，在那里它们被蛋白酶激活。