1.Impairment in the GABAergic system can lead to neuronal hyperexcitability which contributes to ischemia-reperfusion injury.
GABA能系统的异常可致神经元的高兴奋性，从而致再灌注损伤。
2.AIM To observe protective effect of hyperoxia balanced saline solution on reperfusion injury caused by bleeding shock.
目的观察高氧平衡盐液对防治血性休克再灌注损伤效果。
3.This article reviews the progress on the relationship between chronic hypoxia with cerebral hypoperfusion and presenile dementia mechanism.
本文就慢性缺氧/大脑低灌注与早老性痴呆的发机制相关研究的进展作一综述。
4.To investigate the protective effect and mechanism of betaxolol on optic nerves after experimental retinal ischemia-reperfused injury.
探讨倍他洛尔对实验性视网膜缺血再灌注损伤后视神经的保护作用及其可能的作用机制。
5.Mosaic attenuation pattern is a more inclusive term than the original terms mosaic oligemia and perfusion .
该语较原先的语—— 马赛克血量减少和灌注（mosaic oligemia and perfusion）包含更广泛。
6.Measurement Performance in Weak Filling Condition: Oxyhemoglobin and pulse value can be shown correctly when pulse-filling ratio is 0.5%.
6弱灌注情况下的测量性能：在脉搏充盈度为0.5%时能正确显示血氧和脉率值。
7.The RI of cortex was different at various regions, especialy in CGD.The inadiquate perfusion of cortex appeared first at isovolumetric systole.
移植肾皮质部血流灌注不良时，血流充盈缺乏首先发生在等容收缩期。
8.The mechanisms of cognitive impairment caused by carotid diseases are associated with hypoperfusion, leukodystrophy, silent or lacunar infarcts and neuronal degeneration.
颈动脉致认知障碍可能与低灌注、白质变、无症状梗死、腔隙性梗死和神经元变性有关。
9.Objective:To research saluresis types and the weight of deposit in urinary catheter after drug perfusion,as well as the efficacy of drug perfusion.
研究药物灌注后尿路留管内尿盐类型、沉积量及观察药物灌注后的效果。
10.Objective： To investigate the effect of intramyocardial bone marrow cell injection on myocardial perfusion and LV function in patients with chronic myocardial ischemia.
研究骨髓干细胞心肌内注射对慢性心肌缺血患者心肌灌注及左室功能的影响。
11.The paper introduced the construction technology of liquid flyash pouring retailing backwall in order to assure refill quality of retailing backwall,protect retailing backwall depression or jumping.
12.We recorded the changes of the heart rate the blood pressure, and the left ventricle pressure, and observed Ginkgetin effect on reducing the myocardial infarct death rate of AMI - reperfused rabbits.
致神经元的高兴奋性，从而
血性休克再
机制相关研究的进展作一综述。
语较原先的
